Most RP mutations arise in rod photoreceptor genes, leading to diminished peripheral and night vision. To understand why cones die in RP and to be able to stop dying and revitalize dormant cones would be an enormous step forward.
A group of researchers from the University of Louisville has made a step forward into reactivating dormant cones in the animal model of RP. First of all: why cones die in RP? Because rods allow the release of glucose from the Retinal Pigmented Epithelium (RPE) making it available for the cones – rods died, glucose remains trapped inside the RPE, and cones die literally “by starvation”, but before to die they remain for years in a condition named “dormant” – the status between healthy and dead. Dormant is like in a sleeping situation. Researchers have tried on pigs to inject glucose into the affected area, and have seen that not only it restore the normal structure of cones, but also the normal visual function, with a strong increase in photopic ERG. Perhaps sugar can be one of the best neuroprotective agents for cones in RP!
StemActiv, as the best supplement for RP, contains anti-inflammatory, anti-oxidative, anti-apoptotic and neuroprotective substances. It increases glucose supply to the dormant cones and activates the dormant cones. It’s the best timing for RP patients to restore their vision when the optic cells are still in a dormant situation. Nobody can work on the dead cells, it’s impossible to revert blindness when the dormant cells are dead.
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